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33 LOMONTE B. Edema-forming activity of bushmaster Lachesis muta stenophrys ; and Central America rattlesnake Crotalus durissus durissus ; venoms and neutralization by a polyvalent antivenom. Toxicon, 1989, 23, 173-6. LOMONTE B., TARKOWSKI A., HANSON LA. Host response to Bothrops asper snake venom. Analysis of edema formation, inflammatory cells, and cytokine release in a mouse model. Inflammation, 1993, 17, 93-105. MAHMOUD I. Biochemical and physiological studies on the action of viper venom Cerastes vipera on mice Mus musculus. Egypt: Cairo University, Faculty of Science, 1983. 139p. [Masters Thesis] 36 MARKLAND FS., DAMUS PS. Purification and properties of thrombin-like enzyme from the venom of Crotalus adamanteus Eastern diamondback rattlesnake ; . J. Biol. Chem., 1971, 246, 6460-73. MARSH N., GATTULLO D., PAGLIARO P., LOSANO G. The Gaboon viper, Bitis gabonica: haemorrhagic, metabolic, cardiovascular and clinical effects of the venom. Life Sci., 1997, 61, 763-9. MOAV B., MOROZ CH., DE VRIES A. Activation of the fibrinolytic system of the guinea pig following inoculation of Echis colorata venom. Toxicon, 1963, 1, 109-12. MOHAMED A., DAMARAWY NA. The role of the fibrinolytic enzyme system in the haemostatic defects following snake envenomation. Toxicon, 1974, 12, 46775. MOHAMED AH., EL-SEROUGI M., KHALED LZ. Effect of Cerastes cerastes venom on blood coagulation mechanism. Toxicon, 1969, 7, 181-4. MOHAMED AH., KHALED LZ. Effect of the venom of Cerastes cerastes on nerve tissue and skeletal muscle. Toxicon, 1966, 3, 223-4. OUYANG C., TENG CM., CHEN YC. Physicochemical properties of alpha and beta fibrinogenases of Trimeresurus mucrosquamatus venom. Biochim. Biophys. Acta, 1977, 481, 622-30. PERALES J., AMORIM CZ., ROCHA SLG., DOMONT G., MOUSSATCHE H. Neutralization of the oedematogenic activity of Bothrops jararaca venom on the mouse paw by an antibothropic fraction isolated from opossum Didelphis marsupialis ; serum. Agents Actions, 1992, 37, 250-9. REID HA., THEAKSTON RDG. The management of snakebites. Bull. World Health Organ., 1983, 61, 885-95 and isordil.
J. A16015 01 cerebrovascular accident was the cause of her death death certificate of December 29, 1994 ; . The autopsy of 12 28 confirmed this opinion showing that the basilar artery was almost totally obstructed by large atheromatous plaques and recent thrombi with infarction of much of the left hemisphere of the cerebral and cerebellar peduncles, the basal ganglia, brain stem and extending into the cerebellum. It is noted that there was extensive arteriolar atherosclerosis and hyalinization of the kidneys, mild emphysema, fibrosis and acute and chronic bronchitis of the lungs, severe arteriosclerosis of the heart and almost total occlusion of many of the coronary branches with areas of old infarction and fibrosis along the septum and the inferior apical areas as well as hypertrophy of the left ventricle and an aneurysm of the aorta. The summary of the autopsy is that "Following surgery for open repair of a fractured elbow the patient suffered a massive cerebrovascular accident with resultant infarction of most of the left hemisphere of the brain including a portion of the bran stem, cerebellum and basal ganglia area[."] Expert Report of James R. Merikangas, M.D, 10 15 99, at 1-2. 17 Dr. Merikangas clearly stated that, to a reasonable degree of medical certainty, the ischemic event that caused Mrs. Rauch's death was.
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