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P117 AN INTERACTIVE DATABASE FOR GLAUCOMA MANAGEMENT Daniela Paoli Monfalcone Hospital, Gorizia Prov., Italy PURPOSES The author proposes implementation of an Interactive Database System in order to manage medical data about glaucoma patients which can be utilised at a glaucoma centre or a private clinic. METHODS A prototype database is constructed using Microsoft Access software which allows query formulation useful in clinical practice and related research. This system can be expanded and updated. RESULTS Using this system a glaucoma expert can store and analyse data in real time. The system can also be used for statistical analysis. An ophthalmologist who is not an expert in glaucoma can also interact with it to obtain practical advice regarding the patient management in accordance with the EGS guidelines. CONCLUSIONS In the daily practice of glaucoma treatment, an ophthalmologist today deals with a large amount of medical data. High technology diagnostic equipment and pharmacological therapy is able to provide an excellent support for diagnosis and control of glaucoma-based diseases if the use of new data available is also optimal. The idea of a prototype interactive database is proposed which is able to collect and manage such data and where the choice of the treatment among many options is shown visually and in real time. Such a system also provides an opportunity for statistical studies, data storage for follow-ups, and therapy according to EGS guidelines, for example, amitriptyline hydrochloride.
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MATERIALS AND METHODS Enzymes. Restriction endonucleases and T4 DNA ligase were obtained from New England Biolabs, Beverly, Mass. Bacterial strains and plasmids. All of the bacterial strains used were derived from E. coli K-12 and are described in Table 1 or in the text. Plasmids used are described in Table 1, Fig. 3, and the text. Plasmid DNA was prepared as described by Selker et al. 31 ; . Genetic techniques. The techniques used for genetic experiments were as outlined previously 15, 17 ; . Transformations were carried out as described by Lederberg and Cohen 20 ; . TABLE 1. Strains of E. coli and plasmids used and caduet.
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Combinationchemotherapy: Until recently, standard chemotherapy for colorectal cancer usually consisted of treatment with just two drugs, 5-fluorouracil 5-FU ; and leucovorin. A third drug, irinotecan, was approved by the FDA in 1996 for use in combination with 5-FU and leucovorin in treating metastatic colorectal cancer cancer that has spread to other parts of the body ; . Since then, the drug oxaliplatin has also been approved for use in combination with 5-FU and leucovorin to treat metastatic colorectal cancer and post-surgical treatment of this cancer. Unfortunately, traditional chemotherapy agents often affect healthy cells, in addition to cancer cells, leading to a variety of side effects. Monoclonalantibodies: Targeted monoclonal antibody therapies -- bevacizumab Avastin ; and cetuximab Erbitux ; -- have been available since 2004. One advantage of these targeted therapies is that they usually have fewer side effects than chemo drugs. Bevacizumab targets a protein that tumors use to help them grow new blood vessels. The blood vessels let the tumors get the oxygen and nutrients they need to keep growing. Cetuximab targets a protein found on the surface of tumor cells that helps promote cell growth and multiplication. Both antibodies are approved only for people with cancer that has metastasized, and they haven't yet been shown to work in earlier stages of the disease. In addition, bevacizumab may increase the risk of heart attacks and strokes, making it unsafe for certain people.
Table 1.2 Hormonal treatments for menorrhagia and ascorbic, for example, endep sleeping.
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The foam dressing was replaced and the wounds evaluated every 3 to 4 days. Wound dimensions were measured on Days 5, 8, 12, and 36 post injury Table 1 ; . As shown in Figure 5, wound area decreased rapidly between Days 2 initiation of VAC therapy ; and 5, and continued this rapid reduction in size until Day 12. Reduction in wound area was slower between Days 12 and 29 post injury. Nevertheless, wound areas on Day 29 were 29 and 13% of the initial area on the left and right sides of the neck, respectively. Wound depth steadily decreased as granulation tissue filled the defect Table 1; Fig 6 ; . By Day 29 post injury, granulation tissue filled the wounds sufficiently that VAC therapy could be discontinued Fig 7 ; . Aspiration of fluid from the wounds also steadily decreased during treatment. For the first few days, the 1 litre flask required emptying every 10 to 12 the end of the 29-day treatment period, the flask was not full even after 48 h. Light physical therapy was begun on Day 8 with short periods of grazing in hand. On Day 14 the owner began using treats to encourage gentle extension of the horse's neck, with the aim of limiting contracture of the damaged cervical muscles as they healed. Light ridden exercise commenced on Day 29 post injury. Following discontinuation of VAC therapy, wet-to-dry dressings were used to keep the wound surfaces protected until epithelialisation was advanced. The wounds were lavaged with normal saline solution at each dressing change. The owner was instructed to keep the stretch-fabric hood on the horse's neck until epithelialisation of both wounds was complete. In the week following discontinuation of the VAC system between Days 29 and 36 ; , the wounds contracted further and the granulation beds became level with the skin surface Fig 8 ; . Skin grafting was offered at this time but was declined by the client. Despite that, the final functional and cosmetic results were considered very good. The horse had normal mobility of the neck, and the amount of scarring was acceptable to the owner Fig 9 ; . At time during application of VAC wound therapy or following its discontinuation did the jugular vein, oesophagus or trachea appear to be compromised. Neither jugular vein was distended cranial to the injury site, and the horse showed no signs of respiratory embarrassment or difficulty swallowing.
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As outlined in Chapter 1, ZL's approach to expanding HIV care in Haiti is not to provide freestanding, NGO-managed VCT and HIV treatment alone. Rather, the goal is to integrate HIV prevention, testing, and treatment with primary health care in the setting of a public clinic. There are four critical program components--or "pillars"--that serve as entry points for HIV case detection Figure 2.1 ; : 1. people seeking primary health care; 2. people diagnosed with tuberculosis; 3. women seeking women's health or family planning services; and 4. people diagnosed with sexually transmitted infections and chlorthalidone.
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Specific effects as well as peculiar interferences with other steroid hormone signaling pathways, therefore resulting in unique clinical effects. Although these concepts are well established, little research has been devoted to understanding the basis of the differential signaling of progestins. The main finding of this paper is the demonstration that P and MPA trigger significantly different signaling events in controlled in vitro systems. Indeed, we observed that natural P stimulates NO synthesis via transcriptional and nontranscriptional pathways in human endothelial cells as well as in vivo in ovariectomized rat abdominal aorta. In addition, when added to E2, P does not impair the estrogen-dependent induction of eNOS expression, and it even potentiates the effects of estrogen during rapid stimulations. In contrast, MPA does not trigger eNOS expression either in vitro or in vivo, and it does not induce rapid increases of eNOS activity nor does it potentiate E2-dependent nongenomic eNOS activation. Even more interestingly, when prolonged exposures of endothelial cells with E2 and MPA were performed, reductions of estrogendependent eNOS overexpression were seen, implying some sort of interference with estrogen receptor-dependent transcriptional signaling. These effects were confirmed in vivo in ovariectomized rats treated with clinically relevant doses of MPA or P, which supports the possible clinical interest of these results. The clarification of the mechanism of eNOS gene modulation by progestogens is beyond the aim of this paper, but it may be hypothesized that PR, depending on the specific ligand engaging the hormone binding pocket, may be differently able to interfere with transcription factors regulating eNOS expression, such as Sp1 and GATA 35 ; . Indeed, PR has been recently shown to interact with Sp1 in breast cancer cells 36 ; , and up-regulation of GATA-1 has been reported in P-treated erythroid cells 37 ; . Such a mechanism would also.
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Ii - antidepressants, tricyclic systemic ; antidepressants, tricyclic systemic ; some commonly used brand names are: in the anafranil 3 asendin 2 aventyl 7 elavil 1 endep 1 norfranil 6 norpramin 4 pamelor 7 sinequan 5 surmontil 9 tipramine 6 tofranil 6 tofranil-pm 6 vivactil 8 in canada anafranil 3 apo-amitriptyline 1 apo-imipramine 6 apo-trimip 9 asendin 2 aventyl 7 elavil 1 impril 6 levate 1 norpramin 4 novo-doxepin 5 novopramine 6 novo-tripramine 9 novotriptyn 1 pertofrane 4 rhotrimine 9 sinequan 5 surmontil 9 tofranil 6 triadapin 5 triptil 8 note: for quick reference, the following tricyclic antidepressants are numbered to match the corresponding brand names and tenoretic.
The Vascular Disease Foundation and any sponsors disclaim, either explicitly or implicitly, that the drug, biologic, or device listed here is safe or effective for the purposes under investigation, or that the test article is known to be equivalent or superior to any other drug, biologic, or device. Additionally, no claims are made regarding the scientific utility and conduct of clinical trials or research studies listed.
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ENBUCRILATE ENCAINIDE * ENCAPRIN ENCAPSULATED ENCAPSULATION * ENCARE ENCECALIN ENCELIN * ENCEPHABOL ENCEPHALIC $ENCEPHALITIS ENCEPHALITIS-VACCINE ENCEPHALITIS-VIRUS ENCEPHALITOGENIC ENCEPHALITOZOON ENCEPHALOCELE ENCEPHALOGRAPHY ENCEPHALOMALACIA encephalomeningitis ENCEPHALOMYELITIS ENCEPHALOMYELITIS-VACCINE ENCEPHALOMYELITIS-VIRUS encephalomyelitis-virus, equine ENCEPHALOMYELOPATHY ENCEPHALOMYOCARDITIS h.t. h.t. h.t. h.t. use h.t. h.t. h.t. h.t. use h.t. h.t. PROTOZOON ENCEPHALOPATHY HERNIA DIAGNOSIS ENCEPHALOPATHY MENINGOENCEPHALITIS ENCEPHALOPATHY ENCEPHALOPATHY SPINAL-CORD-DISEASE VACCINES PICORNAVIRUS VIRUS EE-VIRUS ENCEPHALOPATHY SPINAL-CORD-DISEASE INFECTION, VIRUS ENCEPHALOPATHY CARDIOPATHY EMC-VIRUS MENTAL-DISORDER NEUROPATHY INFLUENZA-VACCINE h.t. h.t. PSYCHOSEDATIVES TRANQUILIZERS ESTROGEN-ANTAGONISTS PREDNISONE h.t. ANTIDEPRESSANTS PSYCHOSEDATIVES $ENDOCARDITIS ENDOCARDIUM ENDOCERVIX ENDOCHIN * ENDOCISTOBIL ENDOCOCHLEAR * ENDOCORION endocrine-disease use h.t. h.t. h.t. h.t. ENCEPHALOPATHY etc. VACCINES VIRUS ARBOVIRUS ENDOCARDIAL endocardial-fibroelastosis use h.t. h.t. and h.t. h.t. h.t. ENDOCARDIAL LINK FIBROELASTOSIS CARDIOPATHY CARDIOPATHY e.g. INFECTION, BACT. HEART UTERUS PROTOZOACIDES ADIPIODONE MEGLUMINE EAR HCG e.g. ADRENOPATHY THYROID-DISEASE PITUITARY-DISEASE etc. h.t. FUNGICIDES PYRITINOL h.t. PHARMACEUTICS NONOXINOL-9 ENDO-DUPONT * ENDOBIL ENDOBRONCHIAL ENDOBULIN h.t. GLOBULIN IMMUNOGLOBULIN ANTIBODY IODOXAMATE MEGLUMINE h.t. ANTIARRHYTHMICS ASPIRIN ENDARTERITIS ENDEMIC * ENDEP ENDO ENDO-1, 3 4 ; -BETA-D-GLUCANASE h.t. EC-3.2.1.6 ENZYMES DIGESTANTS AMITRIPTYLINE h.t. VASCULAR-DISEASE.
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Second is to reduce the complications caused by the metastases and the weakened bone tissue. But the third and equally important goal is to relieve or prevent pain. Many men with prostate cancer bone metastases experience pain, and dealing with untreated pain expends a lot of energy -- energy that you need to stay healthy and strong. In this Chapter, we'll review some of the more common problems caused by bone metastases as well as the ways that doctors and researchers have learned to treat the metastases and improve the lives of men with advanced prostate cancer and azathioprine.
Athleen Drennan says she "grew up in pharma, " and memories of her first research job conjure a mix of nostalgia and pride. But Drennan, who served on the product development team at Upjohn for 11 years spanning the 1980s and 1990s, also recalls a lack of communication between the R&D group, based in central Kalamazoo, MI, and the promotional division, based miles away in another part of town. "Marketing and R&D were oceans apart, " Drennan says. "We hardly spoke to each other." A decade later, she sees the situation changing. Drennan, who works in clinical recruiting, has seen a rise in the collaboration level between brand teams and medical groups within clients. "Pharmaceutical companies are making great strides bridging that gap between science and marketing, but it's still fragmented, " says Drennan, SVP managing director at Corbett Accel Healthcare Group's Iris Global Clinical Trial Solutions. "Companies are still getting marketing people involved way too late in the game.
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INDICATIONS: MOBAN is indicated for the management ofthe manifestations of psychotic disorders. The anlipsycholic efficacy of MOBAN was established in clinical studies which enrolled newy hospitalized and chronically hospitalized. acutely ill. schizophrensc patients as subjects CONTRAINDICATIONS: Severe central nervous system from any cause, and in patients with known hypersensitivity depression, comatose to the drug. states.
Abbreviations: AR, androgen receptor; FBS, fetal bovine serum; PSA, prostate-specific antigen; TP, testosterone propionate. * To whom reprint requests should be addressed. tThe work described in this paper has been presented in part at the Mary Lasker Symposium: Frontiers in Cancer Research, held by the American Cancer Society, July 28, 1995, San Diego. tA more detailed description of various properties of LNCaP 104-R2 cells will be included in a separate publication by J.K. and S.L. LNCaP 104-R previously reported from this laboratory 6 ; is now designated as LNCaP 104-Ri. LNCaP 104-Ri cells were derived from androgendependent LNCaP 104S cells after 40 passages in DMEM containing charcoal-stripped FBS, whereas LNCaP 104-R2 cells were derived from LNCaP 104-Ri cells after 60 additional passages in the same androgen-depleted medium and co-trimoxazole and endep.
This method of birth control does not protect you from HIV AIDS or other sexually transmitted diseases. Use condoms for increased protection. Don't use this method if: Your periods don't come at a regular time each month, your temperature pattern is different each month, you just stopped birth control pills, you just started having your periods, you are close to menopause, you can't keep a calendar chart, or you don't like going without sex for at least two weeks.
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Omitted from the above table are those ratios that allow the analysis of business risk because ; there was insufficient data submitted and ; the current nature of YES's accounting service does not lend itself to the analysis of business risk. This concept, however, is further explored in Appendix IV as it important factor to be considered when assessing overall current and probable future company performance. To obtain the overall rank, several options were considered. The first option was to add the ranks established for the individual ratios, with the leading company being that with the lowest total. The main difficulty faced with this method was that every ratio could not be calculated for every company and it could have unfairly advantaged those companies who did not have all of the ratios. For example, using this method, a company with no current liabilities would not have a value for the Current Ratio, therefore, having no rank for this ratio. This obviously meant that when adding all of the ranks, the value for this company would be recorded as "n a", which would be ignored by the system and omitted from the set. To get around this problem, the average of the ratios was considered instead. The company with the lowest value at the end of this process was considered to be the leading company in the.
Before initiating or reinstituting HRT, a complete personal and family medical history should be taken. Physical including pelvic and breast ; examination should be guided by this and by the contraindications and warnings for use. During treatment, periodic check-ups are recommended of a frequency and nature adapted to the individual woman. Women should be advised what changes in their breasts should be reported to their doctor or nurse see `Breast cancer' below ; . Investigations, including mammography, should be carried out in accordance with currently accepted screening practices, modified to the clinical needs of the individual. Conditions which need supervision If any of the following conditions are present, have occurred previously, and or have been aggravated during pregnancy or previous hormone treatment, the patient should be closely supervised. It should be taken into account that these conditions may recur or be aggravated during treatment with Totelle Sekvens, in particular: Leiomyoma uterine fibroids ; , or endometriosis A history of, or risk factors for, thromboembolic disorders see below ; . Risk factors for oestrogen dependent tumours, e.g. 1st degree heredity for breast cancer. Hypertension. Liver disorders eg, liver adenoma ; . Diabetes mellitus with or without vascular involvement. Cholelithiasis. Migraine or severe ; headache. Systemic lupus erythematosus. A history of endometrial hyperplasia see below ; . Epilepsy. Asthma. Otosclerosis. Reasons for immediate withdrawal of therapy Therapy should be discontinued in case a contraindication is discovered and in the following situations: Jaundice or deterioration in liver function Significant increase in blood pressure New onset of migraine-type headache Pregnancy Endometrial hyperplasia.
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The Drug-Induced Arrhythmia Risk Evaluation DARE ; study St George's Hospital, University of London and Drug Safety Research Unit, Southampton ; is interested in recruiting patients who have had a proarrhythmic event diagnosed as secondary to therapeutic drug administration or drug overdose. Further details about the study are available from Pauline Telfer, study administrator, on 023 8040 8615 e-mail Pauline.Telfer dsru ; , who can also be contacted by any units interested in a brief presentation about the study.
Data from van Gemert et al. 1998 ; 270 Table 2. Separate variance t-test for differences between the values of the study groups before and after surgery ; and the values of the reference group * p 0.02; * p 0.001; * p 0.001. This facility can be accesses from top View Project Metrics. Then select the desired project from the box captioned Project Name. The organizational metrics screen has five tabs, namely, i. ii. iii. iv. v. Productivity Metrics Defect Metrics Effort Metrics Change Metrics Schedule Metrics.
While implementing fair drug pricing will not hurt the biotech industry, inaction on this item will force the state to cut health budgets and enrollment in health programs, creating a ripple effect that does not end until it hurts taxpayers and the state economy.
Address correspondence to: Dr. Thomas M. Lincoln, Department of Physiology, College of Medicine, University of South Alabama, Mobile, AL. E-mail: tlincoln usouthal, for example, endeo 50.
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Only by consulting a health care provider can you find out for certain whether your child has a urinary tract infection. Some of your child's urine will be collected and examined. The way urine is collected depends on how old your child is. If the child is not yet toilet trained, the health care provider may place a plastic collection bag over your child's genital area. It will be sealed to the skin with an adhesive strip. An older child may be asked to urinate into a container. The sample needs to come as directly into the container as possible to avoid picking up bacteria from the skin or rectal area. A doctor or nurse may need to pass a small tube into the urethra. Urine will drain directly from the bladder into a clean container through this tube called a catheter ; . Sometimes the best way to get the urine is by placing a needle directly into the bladder through the skin of the lower abdomen. Getting urine through the tube or needle will ensure that the urine collected is pure. Some of the urine will be examined under a microscope. If an infection is present, bacteria and sometimes pus will be found in the urine. If the bacteria from the sample are.
638-646. 11. Schwab D, Raithel M, Klein P et al. Immunoglobulin E and eosinophilic cationic protein in , segmental lavage fluid of the small and large bowel identifies patients with food allergy. J Gastroenterol 2001; 96: 508-514. Raithel M, Weidenhiller M, Abel R, et al. Diagnostic use of mucosa oxygenation and histamine release experiments in patients with gastrointestinally mediated allergy GMA ; . Inflamm Res 2003; 52, Suppl 1: S13-S14. 13. Stange EF, Riemann J, von Herbay A et al. Diagnosis and therapy of ulcerative colitis results of an evidence - based consensus conference of the German Society of Digestive and Metabolic Diseases. Z Gastroenterol 2001; 39: 19-20. Brignola C, Campieri M, Bazzocchi G, Farruggia P Tragnone A, Lanfranchi GA. A laboratory , index for predicting relapse in asymptomatic patients with Crohns Disease.Gastroenterology 1986; 91: 1490-1494. Winterkamp S, Weidenhiller M, Wilken V et al. Standardised evaluation of urinary excretion of , N -tele-methylhistamine in different periods of age in a health population. Inflamm Res 2003; 52: S57 - S58 16. Weidenhiller M, Traenkner A, Schwab D, Hahn EG, Raithel M. Different kinetics of mediator release can be detected during allergic reactions after oral provocation double blind placebo controlled food challenge ; . Inflamm Res 2002; 51 Suppl 1: 29-30. 17. Schwab D, Hahn EG, Raithel M. Enhanced histamine metabolism: A comparative analysis of collagenous colitis and food allergy with respect to the role of diet and NSAID use. Inflamm Res 2003; 52 4 ; : 142-147. 18. Werlin SL, Grand RJ. Blody diarrhea -a new complication of sulphasalazine. J Paediatr 1978; 92: 450-451. Williams WR, Pawlowicz A, Davies BH. Aspirin-like effects of selected food additives and industrial sensitizing agents. Clin Exp Allergy 1989; 19: 533-537. Paraskevopoulos I, Konstantinou G. Desensitization treatment of aspirin- and mesalaminesensitive patients with Crohn's disease letter to the editor ; . Inflamm Bowel Dis 2005; 11: 417-418. Turunen U, Elomaa I, Antilla VJ et al. Mesalamine tolerance in patients with inflammatory bowel disease and previous intolerance or allergy to sulphasalazine or sulfonamides. Scand J Gastroenterol l987; 22: 798-802.
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45. Bettuzzi S, Scorcioni F, Astancolle S, Davalli P, Scaltriti M, Corti A 2002 Clusterin SGP-2 ; transient overexpression decreases proliferation rate of SV40-immortalised human prostate epithelial cells by slowing down cell cycle progression. Oncogene 21: 4328-4334 46. Zhou W, Janulis L, Park II, Lee C 2002 A novel anti-proliferative property of clusterin in prostate cancer cells. Life Sci 72: 11-21 47. Bettuzzi S, Zoli M, Ferraguti F, . Ingletti MC, Agnati LF, Corti A 1992 Regional and cellular distribution within the rat prostate of two mRNA species undergoing opposite regulation by androgens. J Endocrinology 132: 361-367 48. Rittmaster RS, Manning AP, Wright AS, Thomas LN, Whitefield S, Norman RW, Lazier CB, Rowden G 1995 Evidence for atrophy and apoptosis in the ventral prostate of rats given the 5 alpha-reductase inhibitor finasteride. Endocrinology 136: 741-8 49. Hsieh TY, Ng CY, Mallouh C, Tazaki H, Wu JM 1996 Regulation of growth, PSA PAP and androgen receptor expression by 1 alpha, 25-dihydroxyvitamin D3 in the androgendependent LNCaP cells. Biochem Biophys Res Commun 223: 141-6 50. Zhao XY, Ly LH, Peehl DM, Feldman D 1997 1alpha, 25-dihydroxyvitamin D3 actions in LNCaP human prostate cancer cells are androgen-dependent. Endocrinology 138: 3290-8 51. McGuire TF, Trump DL, Johnson CS 2001 Vitamin D 3 ; -induced apoptosis of murine squamous cell carcinoma cells. Selective induction of caspase-dependent MEK cleavage and up-regulation of MEKK-1. J Biol Chem 276: 26365-73 52. Bernardi RJ, Trump DL, Yu WD, McGuire TF, Hershberger PA, Johnson CS 2001 Combination of 1alpha, 25-dihydroxyvitamin D 3 ; with dexamethasone enhances cell cycle arrest and apoptosis: role of nuclear receptor cross-talk and Erk Akt signaling. Clin Cancer Res 7: 4164-73 53. Bettoun DJ, Buck DW 2nd, Lu J, Khalifa B, Chin WW, Nagpal S 2002 A vitamin D receptor-Ser Thr phosphatase-p70 S6 kinase complex and modulation of its enzymatic activities by the ligand. J Biol Chem 277: 24847-50 26.
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